记者12日从武汉大学基础医学院获悉，该院年轻教授侯炜和武汉大学动物实验中心科研人员合作的一项科研成果发表在8月份美国著名国际学术刊物《淋巴细胞生物学》杂志上。这项研究发现，一种名为“表达CD56分子的T淋巴细胞”具有抗艾滋病毒感染的作用。

Reporter 12 from wuhan university of basic medical college,The hospital of wuhan university professor young HouHui and animal experimental center of scientific research personnel cooperation a scientific research published in the August the famous American international academic journals[T cell biology]magazine.The study found that,A name"Express CD56 molecular T lymphocyte"It can resist the effect of HIV infection.

　　据侯炜教授介绍，这种名为“表达CD56分子的T淋巴细胞”是一类“桥梁细胞”，既具有自然杀伤功能，又能起到自我保护作用。它是人类天然免疫系统的一个重要的组成部分，在外周血单核细胞中占5%-15%，而肠道和肝脏中的50%免疫细胞为“表达CD56分子的T淋巴细胞”。此前研究只被发现这种细胞在抗肿瘤和抗肝炎病毒感染中起作用，而没有发现它在抗艾滋病毒感染有较强作用。

According to HouHui introduced professor,This name"Express CD56 molecular T lymphocyte"Is a kind of"Bridge cell",Not only has the function of natural killer,And can play the self protection.It is human natural immune system an important part,In the peripheral blood mononuclear cells 5% - 15%,And the intestinal and liver of 50% for immune cells"Express CD56 molecular T lymphocyte".Previous studies is found that the cells in antitumor and anti hepatitis virus infection play a role,But didn't find it in the anti HIV infection have a stronger role.

　　侯炜教授和武汉大学动物实验中心霍文哲教授团队历经3年多研究，首次发现“表达CD56分子的T淋巴细胞”培养液中的分泌物可以抑制艾滋病毒的感染和复制，并且这种活性具有广谱性，既可抑制实验室保存的艾滋病毒病毒株，也可抑制临床上分离得到的艾滋病毒病毒株。

HouHui professors and wuhan university animal experiment center HuoWenZhe professor team after 3 years research,First discovered"Express CD56 molecular T lymphocyte"Nutrient solution of secretion can inhibit infection with the AIDS virus and copy,And this kind of activity have a broad spectrum of sex,Can inhibit laboratory saved the AIDS virus strains,Also can inhibit clinically separation get AIDS virus strains.

　　这个研究团队发现，虽然该培养液中的分泌物对艾滋病毒进入细胞的协同受体影响甚微，但可增强干扰素调节因子的作用，从而引起巨噬细胞发挥作用，“抗击”艾滋病毒。(记者李鹏翔)

The research team found,Although the secretion of nutrient solution to the AIDS virus into the cells' cooperative receptor has little effect,But can enhance the effect of interferon regulatory factor,Thus cause macrophages play a role,"fight"HIV.(Reporter pengxiang li)